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Long-term hypoxia and inflammation in the lipid core cause foam cells to die in a variety of ways, including necrosis and apoptosis

Complete tears where the tendon has fully separated from bone

By regulating arsenic sequestration mechanisms, JA reduces cytosolic As levels, compartmentalizing it into vacuoles and cell walls, thus preventing its interference with essential metabolic functions 51

3 Dual targeting for optimal treatment of autoimmune diseases Early in the disease course, CD19-based therapy may prevent autoreactive plasma cell accumulation, but later, memory plasma cells may accumulate and lead to persistent autoantibody production despite B-cell depletion (32, 33)

[DOI] [PubMed] [Google Scholar] Elamin, A