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By targeting inflammation and senescent cells, we may disrupt chronic disease cycle and unlock new preventive and therapeutic possibilities for oral and systemic health. Cells exposed to stressors like inflammation, oxidative stress, and microbial toxins can enter a senescent state, ceasing to divide and losing normal function

It oxidizes into glutathione disulfide (GSSG), where the SS denotes the disulfide bond between two glutathiones

[PubMed ID: 36312537]

Abstract Cellular senescence, driven by the interaction between FOXO4 and p53, is increasingly recognized as a crucial mechanism in brain aging and the development of neurodegenerative disorders

In addition, we observed an increase in the percentage of cells in both early and late apoptosis after BSO treatment, suggesting that full GSH content is necessary for proliferation and survival of CSCs (Figure 4C)