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EMF exposure has been documented to reduce ATP production and cause mitochondrial fragmentation following chronic exposure. This is the mechanism behind fatigue that does not respond to sleep, post-exertional crashes, and cognitive impairment persisting for days after significant EMF exposure

Furthermore, the terminal amine of these precursors can be conjugated with functional groups that respond to specific stimuli such as light, redox-reactions, or enzymes to achieve spatiotemporal control over RSSH release

[PMC free article: PMC4330973] [PubMed: 24831770] 166

and minerals, such as calcium and iron

J Dermatolog Treat 2012;23(2):97-102