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Description
doi: 10.1016/j.jep.2021.114503
Brinzolamide induced reversible corneal decompensation
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Overall, the resistance to ferroptosis of GPX4 requires catalytic selenocysteine residues and depletion of GSH, and inhibition of peroxide accumulation is achieved by GSR/GSH (glutathione disulfide reductase) and NADPH/H+ recirculation
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Time to onset of symptoms and lab abnormalities varied from 14 to 540 days
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