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Therefore, high pro-inflammatory cytokines and activated TLR4 in Covid-19 may lead to activation release of PRL from anterior pituitary and could explain hyperprolactinemia in Covid-19

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In addition, oxidative damage is likely to be evoked as one of the main pathways of ZEA toxicity which may initiate event at least in part contribute to the mechanism of ZEA induced genotoxic and cytotoxic effects [126]