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The hormone is associated with the regulation of physiological functions, such as the sleep cycle, immune function, homeostasis, and glucose regulation [12, 13], and is beneficial to the cardiovascular system [14]

Although epigenetic monotherapies such as low-dose DNA methyltransferase inhibitors (azacitidine/decitabine) and bromodomain, have demonstrated the ability to eradicate chemotherapy-induced senescent AML subsets, deplete leukemic stem-like cells, and enhance initial responses in refractory AML, their efficacy as stand-alone regimens is significantly limited

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In particular, lack of early response appears to be strongly indicative of subsequent nonresponse [2], failure to achieve remission [3], and higher rates of treatment discontinuation [4]

Several shortened variants have been described which presumably arise by differential processing of the precursor protein