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F.ZouT.ShuY.et al (2022)
Some of the areas of research addressed in recent scientific literature include dentist and dental staff occupational exposure to mercury [11] [12] [13] [14] [15] [16] [17] [18] [19] [20] [21] [22] [23] [24] [25] [26] [27] [28] [29] [30] [31] [32] [33] [34] [35] [36] [37] [38] [39] [40] [41] mercury vapor releases from amalgam fillings [42] [43] [44] [45] [46] [47] [48] [49] [50] [51] [52] [53] [54] [55] [56] [57] [58] [59] [60] [61] [62] [63] mercury vapor releases increased by the number of fillings, chewing, teeth-grinding, brushing, dental treatments, and hot liquids [64] [65] [66] [67] [68] [69] [70] [71] [72] [73] genetic traits impacting response to dental mercury [74] [75] [76] [77] [78] [79] [80] fetal and infant exposure to mercury via maternal dental mercury amalgam [81] [82] [83] [84] [85] [86] [87] [88] [89] [90] [91] [92] [93] [94] [95] [96] mercury excreted in breast milk of mothers with dental mercury amalgam fillings, increasing with the number of amalgam fillings [97] [98] [99] [100] dangers of dental mercury use in children [101] [102] [103] [104] [105] [106] [107] [108] [109] [110] allergy/hypersensitivity to dental mercury [111] [112] [113] [114] [115] [116] [117] [118] [119] [120] [121] [122] [123] [124] [125] [126] [127] [128] dental amalgam mercurys impact on patients with autoimmune disorders/immunodeficiency [129] [130] [131] [132] [133] [134] [135] dental amalgam mercurys impact on patients with kidney disease [136] [137] [138] [139] [140] dental amalgam mercurys impact on patients with multiple sclerosis [141] [142] [143] [144] dental amalgam mercurys impact on patients with oral lichenoid reaction [145] [146] [147] [148] [149] [150] [151] and oral lichen planus [152] [153] [154] Dental amalgam controversy: worldwide regulations begin in the 21st Century Awareness of occupational hazards, environmental dangers, and patient risks as a result of dental mercury usage has grown with new science

A well-balanced equilibrium between mitochondrial ROS production and their elimination is critical for the control of redox-mediated cell signalling processes 1

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