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Our results from experiments using siRNA-mediated knockdowns of STAT3 and GIV suggest that the effects of IL-17 were mediated by activating STAT3/GIV signaling in NSCLC cells and subsequently up-regulating its downstream target VEGF
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Liver diseases, particularly non-alcoholic fatty liver disease (NAFLD) and metabolic dysfunction-associated steatotic liver disease (MASLD), have grown to be...
Finally, we discuss successful outcomes of therapeutics that target S-nitrosylation of proteins in Parkinsons Disease related clinical trials
Biomed Res Int
