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Restricting substrate influx (e.g., fatty acids or succinate) avoids over-reducing the electron carriers at the critical moment of reperfusion, and specifically blunting reverse electron transport into Complex I limits the high-amplitude ROS spikes that drive acute injury and propagate inflammatory signaling

Dual Fluorescence Nanoconjugates for Ratiometric Detection of Reactive Oxygen Species in Inflammatory Cells

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In response to this activation signal, AIM2 or NLRP3 associate with ASC and pro-caspase 1 to form the active inflammasome, a multiprotein complex that liberates active caspase-1 to facilitate the maturation of IL-1 and IL-18