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During development and progression of atherosclerosis, Nrf2 signaling modulates many physiological and pathophysiological processes, like regulation of lipid homeostasis, CD36 gene expression regulation, foam cell formation, macrophage polarization, immunity regulation (Th2 differentiation and inhibition of pro-inflammatory gene expression through NFB down-regulation), redox regulation and inflammation, improvement of endothelial dysfunction, as well as GSH synthesis and utilization (234245)

Most critically, NAC strongly potentiates the effects of nitroglycerin and other nitrate vasodilators

The results showed that F1 was cleaved after Phe-132 and F2 was cleaved after Phe-235, both of which are in line with the catalytic activity of PrtP as a phenylalanine protease (Fenno et al, 2001