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Collected studies from our groups and others have contributed to unraveling the close relationships between perturbation of redox homeostasis in the brain and AD neuropathology by elucidating redox-regulated events potentially involved in both the pathogenesis and progression of AD

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This strategy is more relevant for enhancing treatment with DNA-damaging chemotherapeutics than with targeted therapeutics, as the chemotherapeutic treatments generally lead to SASP-induction 47

thirdly, the detailed changes of the downstream of the activated SNr GAD2 neurons in HE are not clarified