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There are several lines of evidence correlating the depletion of intracellular glutathione with generation of ROS/RNS and progression of apoptosis in SLE patients [50,61,78]

The mechanistic and in-vitro evidence is strong and well-replicated

Xing L, Shieh HS, Selness SR, Devraj RV, Walker JK, Devadas B, et al

[30] Release of GSTs as an indication of organ damage [edit] The high intracellular concentrations of GSTs coupled with their cell-specific cellular distribution allows them to function as biomarkers for localising and monitoring injury to defined cell types

Mechanistically, GHK-Cu preserved mitochondrial function by increasing mitochondrial membrane potential, alleviating age-related mitochondrial network fragmentation, shifting mitochondrial dynamics toward fusion via regulating drp-1 and fzo-1 expression, and promoting ATP biosynthesis