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Analysis of CTCs obtained from patients and mouse models during the resting phase using scRNA-seq reveals significant upregulation of mitotic genes, which may contribute to the enhanced metastatic potential of CTCs

Agrillo A, Ungari C, Filiaci F, Priore P, Iannetti G

Figure 4A shows the enriched pathways identified separately for ELCs, RLCs and LLCs, with color indicating the enrichment score, and the size of each dot corresponding to the number of transcripts in that cluster

This dual action, combining enzymatic cofactor delivery with transcriptional modulation, underpins GHK-Cu's unusually wide range of documented biological effects