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Description
The portion of ALCAR that does survive intact is uniquely capable of crossing the blood-brain barrier, a feat that LCT cannot achieve
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Transient transfections were performed using a wild-type or mutated (to disrupt the consensus DR-1 sequence at 70 bp from the start site of transcription) human SHBG promoter-driven luciferase reporter plasmids together with a pCMRenilla as control plasmid in the presence or absence of hCAR or mCAR expression vectors
What wrong with the Kappa Betas
Methods Pathol
