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Thus, modulating astrocytic autophagy represents a potential therapeutic strategy, provided that the intervention can restore equilibrium rather than simply suppressing or hyperactivating the pathway [57-60]

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In IPF, GSH levels are significantly reduced, contributing to a pro-oxidant environment that promotes fibroblast proliferation and ECM accumulation [120]

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i n vitro and in vivo studies done so far underscore the chances of ADE in Flaviviruses infection

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