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Jpn J Pharmacol 1993

But you may be surprised to discover that this isnt necessarily the case

Zinc deficiency in these organelles appears to be associated with oxidative stress pathways, including the phosphorylation of initiation factor eukaryotic 4, the increased expression of C/EBP, the release of cytochrome c, and the insertion of Bax, a protein of the Bcl-2 family which activates cell death, with subsequent activation of caspase-3 and apoptosis [43]

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They achieve this by reducing the levels of MDA and reactive oxygen intermediates and increasing the activities of SOD and GPX (Arat et al., 2010