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Sam and Bordoni, 2022)

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As part of its response to inflammation, the body can activate oxidative stress resistance mechanisms (such as those centered around the transcription factor NRF2) in addition to inducing inflammatory mechanisms ( Sex-dimorphic regulation of ferroptosis in atherosclerosis Atherosclerosis exhibits well-documented sex differences in incidence, progression, and plaque phenotype, with young women being relatively protected compared to age-matched men ( - / - mice, estrogen deficiency accelerates atherosclerosis with increased lipid peroxidation and iron deposition

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