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Perturbations in peroxisomal activity that disturb redox homeostasis are implicated in the carcinogenesis of prostate cancer and hematological malignancy [18]

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We also observed a significantly steady decline of median CQ IC 50 from a high of 92.8 nM, n = 49 (IQR 39.5 nM to 163.3 nM) in 2008 to a low of 22.4 nM, n = 53 (IQR