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Funding This research was supported partially by the NIH (MH093458, MH113645, and AG079129), Johns Hopkins Medicine Discovery Fund, and UAB start-up funds to S-IK

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Given EHMT2/G9as role in inflammatory regulation and KL1s impact on ROS/RNS-related gene expression, we hypothesized that KL1 facilitates antibiotic killing by reducing ROS/RNS production
