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The mechanisms studied in animal models include growing new blood vessels (angiogenesis, via VEGFR2), helping repair cells move to the injury (fibroblast migration, via the FAK-paxillin pathway), and supporting blood flow through the nitric-oxide pathway

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Suppression of NRF2-mediated cytoprotective signaling enhanced cisplatin-induced DNA damage and apoptosis, as demonstrated by increased levels of cleaved caspase-3 and PARP in vitro, along with a pronounced restoration of drug sensitivity in vivo

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