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Therefore, the present study investigated whether BSO induced ferroptosis in cancer cells, and whether the cellular glutathione level may be a marker for GCLC inhibitor-sensitivity

In addition, the subsequent immunohistochemical analysis demonstrated that the treatment with both isothiocyanates reduced the loss of dopaminergic neurons and apoptosis [92] (see Table 3)

This effect was reversed by co-expression of the wild-type PGRMC1 but not of the Y113F mutant, suggesting that PGRMC1 enhances doxorubicin resistance of cancer cells by facilitating its degradation via cytochromes P450

L Hagens, Stockholm

Mold toxins must be detoxified through the liver and the cytochrome P450 pathway