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and anemia NOS (D64.9), offering a versatile framework for coding different manifestations of macrocytic anemia

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GSNO may be animportant physiological NO donor, for example, in the regulation of NPR1, where nitrosylation of proteine cysteine residues acts antagonistically to reduction by TRX (Tada et al., 2008)

This study demonstrated that EZH2 was significantly upregulated in kidney stone patients, kidney stone mice, and oxalate-stimulated HK-2 cells