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Despite significant progress in understanding ferroptosis in AS and depression, its role in comorbid conditions remains unclear

Levels of homocysteine may be increased by dietary habits, prescription medicines, or enzymatic mutations affecting homocysteine metabolism.43The results of meta-analyses confirm total homocysteine to be an independent risk factor for RVO.21,44Loewenstein et al.45 investigated the prevalence of genetic mutation in the enzyme methylentetrahydrofolate reductase (MTHFR) whose impaired activity may lead to hyperhomocysteinemia

Incomplete or insufficient response: Failure to achieve remission characterized by the lack of normalization of transaminases and immunoglobulin G levels within 6 months despite adherence to therapy