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One possibility is that changes in miR203-5p (and/or other miRNA molecules not examined in the present study) lead to transcriptional changes, potentially through other epigenetic mechanisms such as histone methylation/acetylation and heterochromatin modifications, that escalate over time and result in impaired glutamate homeostasis and glutamate-GABA communication

AMPK inhibits mTOR, and downregulation of mTOR has been shown to promote lifespan in several organisms and thus regulation of mTOR by AMPK provides another likely mechanism by which AMPK could influence lifespan (Jia et al., 2004