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Studies from this group support that Pb can disrupt the brain export of A leading to its accumulation and plaques formation (Behl et al., Exposure to Pb during development is a good example of an environmental contaminant as a risk factor to promote neurodegenerative diseases such as AD, supporting the hypothesis that many adult diseases have a fetal origin (FeBAD) (Basha et al., APP mRNA expression as well as in the activity of the transcription factor Sp1 (one of the regulators of APP ) in the cortex of neonates, and after 20 months of Pb exposure had ceased

doi: 10.1016/j.jsps.2018.11.007 46 SantosAMLippoBSBahrkeMSYonamineM

These molecules are used clinically or experimentally to reduce oxidative damage, particularly in conditions like neurodegenerative disorders, ischemiareperfusion injury, and inflammation-related diseases [100]