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Comprehensive clinical assessment identifies specific neurocognitive deficits in working-age patients with long-COVID

In primary cultures of cortical neurons, it was observed that increased lentiviral-mediated Pfkfb3 expression resulted in a limited but significant stimulation of glucose utilization (20%) (Blanger et al., 2)
Depletion of glutathione (GSH), mitochondrial damage and oxidative stress are key factors in the development of acetaminophen (APAP) hepatotoxicity (74-76)

Increased levels of systemic LPS-positive bacterial extracellular vesicles in patients with intestinal barrier dysfunction

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