glutathione s transferase gene Glutathione-S-transferase interacting network. We utilized the IPA Glutathione: Master Antioxidant, Reducing Oxidative

In vitro experiments demonstrated that treating primary mouse lung fibroblasts stimulated by TGF-1 with piceid can reduce LOX2 expression

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Its primary mechanisms are: Actin regulation critical for cell migration and tissue repair Anti-inflammatory signalling through the NF-B pathway19 Angiogenesis promoting new vessel formation in damaged tissue20 Cardioprotection demonstrating direct myocardial repair properties in animal models21 TB-500 Cardiovascular Long COVID: The Strongest Use Case Long COVID cardiovascular sequelae are well-documented: Myocarditis and pericarditis in a subset of patients POTS (postural orthostatic tachycardia syndrome) Reduced VO max and exercise intolerance Microclotting and endothelial damage TB-500's most compelling evidence base is cardiovascular

Attia et al., 2019

Will have to reread it a couple of times