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however, recent findings indicate that amyloid formation in vivo may be promoted by disulfide bond formation between TTR subunits, suggesting oxidative stress as a potential contributor to protein misfolding and disease progression
Epigenetics 8, 730-738
10.1104/pp.002337 Plant Physiol
268 In AD-like tauopathies, HDAC7 suppresses lysosomal biogenesis by deacetylating TFEB, thereby inhibiting its nuclear translocation and reducing the expression of genes such as LAMP2 and CTSD
Mitochondria, as central regulators of energy metabolism and apoptosis, play a critical role in sepsis-induced intestinal barrier injury
