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The acquisition of a memory phenotype and the recovery of an efficient CD8 + T cell function declined with HCV infections PD-1 expression, whereas, when HCV persisted and the HCV-specific CD8 + cells remained dysfunctional, high levels of PD-1 were maintained, the PD-1 ligation likely provides an overall inhibitory signal to Tregs cells shown in a study of PD-1 expression on Tregs in humans during the chronic HCV infection

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