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Since hsp-90 is a well-known target of HSF-1, it is conceivable that depleting HSF-1 may impair the proper induction of the UPR ER upon heat- or tunicamycin-induced stress due to insufficient HSP-90 levels
Monthly monitoring supports dose tolerance assessment
2017;16:523-541.[PubMed Abstract] - 102.Michot JM, Canioni D, Driss H, et al
