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In CRC, treatment with catalpol reduced autophagy, triggered apoptosis, decreased cell viability, and regulated SIRT1 expression by increasing miR-34a levels both in-vivo and in-vitro
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Specifically, GCLC expression fell during HSC activation and increased as activated HSCs revert to quiescence
Influence of molecular structure of astaxanthin esters on their stability and bioavailability
