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It neutralizes reactive species such as singlet oxygen and prevents lipid and protein oxidation, demonstrating stronger protective effects than nicotinic acid 86
doi: 10.1016/j.abb.2004.09.024 170
The cleavage of N-terminal domain forms membrane pores, leading to cell lysis and the release of interleukin (IL)-1 and IL-18, which amplify immune responses [10]
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IRAK4 primes NOX and phosphorylates p47phox on several residues to activate the oxidase directly [251]
