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Drugs Aging (2004) 21:37793.10.2165/00002512-200421060-00003 7 IwanamiUMogiMIwaiMHoriuchiM

Despite promising effects of GluN2B antagonists in HD and other NMDA excitotoxic disease models, there are still limitations, namely 1) potential inefficacy of GluN2B antagonism without additional potentiation of GluN2A and subsequent cell survival signaling

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In vitro tracing of 13 C-labelled glucose and BHB Splenic mouse CD4 + T cells were activated in culture for three days