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The epithelial-to-mesenchymal transition (EMT) is the process by which epithelial cells lose their junctions and apical-basal polarity, and then increase the mobility of single cells and make the development of invasive phenotype possible [110]

The authors reported that co-treatment with N-acetylcysteine (NAC) partially rescued cell viability in both Mirk-depleted and control cells treated with cisplatin, underscoring the critical role of redox modulation in Mirk-targeted strategies

145,146,147 Currently, it is widely believed that BMAL1 exerts its influence on angiogenesis through the regulation of VEGF

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