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Recent evidence has revealed mitochondrial dysfunction in RA PBMCs, involving reduced ATP, disrupted m, elevated ROS, and upregulated ROMO1/OMA1 expression ( 2 O 2 -induced) upregulates MST1 expression in RA-FLSs, triggering mitochondrial dysfunction and apoptosis ( Taken together, mitochondrial metabolism reprogramming not only plays a key role in RA pathophysiology, but may be a promising potential target for future treatment of RA

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However, there is strong evidence supporting the connection between oxidative stress and age-related disease development

To test the short-term temporal dynamics of methylation and demethylation effects, we measured the expressions of epigenetic modifying enzyme genes in BSO treatment withdrawal conditions in an in vitro mouse hepatocyte model

Recent research shows that the gut microbiome may affect the course of ALS (7)