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doi: 10.1016/j.ophtha.2008.05.015

This disruption prevents metabolic reprogramming toward an effector phenotype, inhibiting Teff cell differentiation while potentially favoring Treg generation

These findings further ruled out mitochondrial damage to be the source of GSH depletion-induced oxidative stress
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We further thank the UK Biobank Resource, with the replication analysis in this study having been conducted under application number 95801
