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Regarding the prevailing view that the SEC acts as a positive cofactor for stress-induced genes, 6KR MED1-mediated effects on stress-inhibited genes suggest either context-dependent SEC repression or SEC-independent regulation through altered Pol II recruitmenta dichotomy requiring further investigation
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Instead, it may offer slow, consistent reinforcement of the body's natural repair and recovery mechanisms
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We next examined the potential of Immunocal to ameliorate damage induced by excitotoxic insult in NSC34 cells, which were differentiated by prolonged serum withdrawal to induce the expression of NMDA receptors [47]
