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MicroRNA-98: the multifaceted regulator in human cancer progression and therapy

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Various studies from epithelial monolayers reported that PM led to increases in permeability of nasal (43), bronchial (46), and alveolar epithelial cell cultures (47) that was associated with disrupted tight junctions, downregulation of tight junction proteins such as ZO-1, occludin, claudin1, and E-cadherin ( Figure 1 )

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