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Description
The use of MET inhibitors may disrupt typical downstream pathways, specifically JAK/STAT1, which are common to both MET and IFN-, thereby impeding the IFN--triggered expression of PD-L1 and PD-L2 in tumors with MET amplification

doi:10.1016/0167-4838(89)90159-3
Riddle, E

[20] Both subunits of the GST dimer, whether hetero- or homodimeric in nature, contain a single nonsubstrate binding site, as well as a GSH-binding site

Health promotion is one of the most essential instruments of infectious disease management
