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Conclusions In summary, our study evidenced that AT-specific B12 deficiency significantly dysregulated AT lipid metabolism and inflammation, thereby promoting dyslipidemia and worsening obesity-related complications that could underlie the development of IR and cardiometabolic risk

The central application of neurotensin as a neurotransmitter was a central basis of its cytoprotective effect but was not further extended [151]

Storage Conditions and Notes Liquid and vapors are flammable