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Description
This work was supported by grants from the Department of Science and Technology, Govt
Extensive research has indicated that the HGF-cMet pathway protects against oxidative stress by upregulating antioxidant proteins like superoxide dismutase and catalase and stimulating GSH biosynthesis (Gomez-Quiroz et al
These studies have identified candidate biomarkers and distinct pathophysiologic signatures, which may inform disease classification, risk stratification, and therapeutic targeting [2529]
The findings demonstrated that CUR@DssD-Hb NPs and CUR/miR155@DssD-Hb NPs substantially augmented the secretion of HMGB1 and ATP
doi: 10.1002/med.21360
