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Description
Glutamine synthetase (GS) can maintain the phenotype of M2 macrophages, and the inhibition of GS skews M2-polarized macrophages toward the M1-like phenotype [27]
doi: 10.1016/j.freeradbiomed.2007.03.024 [DOI] [PMC free article] [PubMed] [Google Scholar] 93
In FLT3-ITD-positive AML, ATF4 activation has been discovered to be intimately linked with the proliferation of autophagy-dependent AML cells
For example, long-term supplementation might be advised If you have a glutathione deficiency or related disease
PMID: 29156024
