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glutathione breast milk copper excess induces autophagy dysfunction and mitochondrial ROS-ferroptosis progression, inhibits cellular biosynthesis of protein and lipid in bovine mammary epithelial cells The physiological role of copper:

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12 emerges as key player during cellular reprogramming"

glutathione breast milk copper excess induces autophagy dysfunction and mitochondrial ROS-ferroptosis progression, inhibits cellular biosynthesis of protein and lipid in bovine mammary epithelial cells The physiological role of copper:

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glutathione breast milk copper excess induces autophagy dysfunction and mitochondrial ROS-ferroptosis progression, inhibits cellular biosynthesis of protein and lipid in bovine mammary epithelial cells The physiological role of copper:

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glutathione breast milk copper excess induces autophagy dysfunction and mitochondrial ROS-ferroptosis progression, inhibits cellular biosynthesis of protein and lipid in bovine mammary epithelial cells The physiological role of copper:

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glutathione breast milk copper excess induces autophagy dysfunction and mitochondrial ROS-ferroptosis progression, inhibits cellular biosynthesis of protein and lipid in bovine mammary epithelial cells The physiological role of copper:

*Correspondence: Yanfang Liu, [email protected] Wang, [email protected] Zhu, [email protected] These authors have contributed equally to this work Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

glutathione breast milk copper excess induces autophagy dysfunction and mitochondrial ROS-ferroptosis progression, inhibits cellular biosynthesis of protein and lipid in bovine mammary epithelial cells The physiological role of copper:

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