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The molecular docking results revealed that the VPA and CBZ compounds could interact with the identified targets active sites, and thus, the identified proteins may be suited for oxidative stress mediated therapy in epilepsy

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When DHHC13-mediated S-palmitoylation of ULK1 enhances its recruitment to autophagosome formation sites (Tabata et al, 2024), and DHHC5-mediated S-palmitoylation of Beclin-1 promotes class III phosphatidylinositol-3-kinase complex assembly (Guo et al, 2024), ZDHHC7-mediated S-palmitoylation of ATG16L1 facilitates LC3 lipidation during autophagy (Wei et al, 2024)