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The OSC-induced changes in the proliferation of cancer cells are frequently associated with perturbations in cell cycle progression and induction of G2/M phase arrest.[5] The OSC induces apoptosis via the intrinsic pathway by altering the ratio of the Bcl-2 family of proteins

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Sechi et al., Progress in Neuropsychopharmacology & Biological Psychiatry, 10/96 A review of the evidence concerning hepatic glutathione depletion and susceptibility to hepatotoxicity after Paracetamol overdose by S.S