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Furthermore, in a rat model of OA induced by mono-iodoacetate (MIA), B2A treatment significantly enhanced the quality of cartilage, as evidenced by increased GAG production and a higher density of cartilaginous cells

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Most of these conditions are painful inflammation, fluid accumulation, or require direct medication delivery to the joint or bursa
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MOTS-c activates AMPK, which signals the body to burn existing fat stores
