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The ability of TST to modulate sulfide metabolism, interact with key metabolic pathways and exert anti-oxidative effects underscores its significance in both preclinical models and potential clinical applications for conditions related to those biological processes

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Elevated intracellular glucose levels have been shown to induce stress-related gene expression, suggesting a potential mechanistic interaction between sugar signaling and SL pathways, particularly evident in Hxk1 and Max2 mutants 182
