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[DOI] [PubMed] [Google Scholar] 66.Molteni A., Nickerson P.A., Brownie A.C., Liu K

DAMPs such as HMGB1, IL-33, and ATP released from liver injury bind to TLR2/4 and activate hepatic stellate cells (HSC), prompting their transformation into myofibroblasts and secretion of a large number of extracellular matrix proteins, which leads to matrix deposition and fibrosis [277, 316, 317]

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Following this, elucidation of a solid mechanism by which chronic metabolically induced ROS triggers the progression of metabolic disease pathology in organs such as the liver, kidney, and heart would be required