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[9] It is hypothesized that GHK binds to ferritin channels, possibly reducing iron release by as much as 87%, which may play a role in mitigating further oxidative damage and inflammation in compromised tissues

In terms of mechanism, DNA damage repair is the most critical reason for the radiotherapy resistance of GH and IGF1 in cancer cells and damage repair effect on adjacent tissues

Each problem requires a different approach

G., Moxley, R

doi: 10.1016/j.nefro.2015.05.022 126
