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[ There is still a controversy whether high UA is a compensatory mechanism to overcome increased oxidative stress, which is an independent cause of cardiovascular disease [ and is a component of the inflammatory process
The molecular docking results for luteolin are presented in Table 4

IGF1 stimulation could induce DNA repair by HR in MCF7 cells (Trojanek et al., 2003

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2017;26:509e5226
